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IL-17A is Elevated in End-stage COPD and Contributes to Cigarette Smoke-induced Lymphoid Neogenesis.

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Author

Summary, in English

End-stage chronic obstructive pulmonary disease (COPD) is associated with an accumulation of pulmonary lymphoid follicles. Interleukin (IL)-17A is implicated in COPD and pulmonary lymphoid neogenesis in response to microbial stimuli. We hypothesized that IL-17A is increased in peripheral lung tissue during end-stage COPD and also directly contributes to cigarette smoke-induced lymphoid neogenesis.

Publishing year

2015

Language

English

Pages

1232-1241

Publication/Series

American Journal of Respiratory and Critical Care Medicine

Volume

191

Issue

11

Document type

Journal article

Publisher

American Thoracic Society

Topic

  • Respiratory Medicine and Allergy

Status

Published

Research group

  • Airway Inflammation and Immunology

ISBN/ISSN/Other

  • ISSN: 1535-4970